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Exploring the Potential Link Between COVID-19 Vaccines and Kidney Damage: A Personal Account

After recovering from COVID-19 in September 2020 with normal kidney function, my health sharply declined following two doses of the Pfizer mRNA vaccine. I developed dysautonomia, congestive heart failure, severe neuropathy, and rapidly worsening kidney function that led to dialysis. Initially, I believed diabetes caused my kidney failure, but the sudden progression did not match typical diabetic patterns. Testing years later revealed persistent spike protein in my body even five years post-vaccination, linking my condition to vaccine-related injury. My story highlights the urgent need for awareness, acknowledgment, and support for those suffering long-term, life-altering complications. Tags: #DialysisJourney #VaccineInjury #SpikeProteinPersistence #CIDP #ChronicIllnessAwareness #KidneyFailure #MedicalJustice

Glenn Rosaroso Vale, MT(AMT), MS(IT), MBA

9/1/20252 min read

How the COVID-19 Vaccine Could Lead to Kidney Damage and Dialysis: A Personal Perspective

Since the rollout of COVID-19 vaccines in 2020, reports of unexpected health complications, including kidney damage, have started to surface. While vaccines are intended to protect against viral infections according to government, emerging evidence suggests they may trigger mechanisms that stress or damage vital organs. Here’s a deeper look at how the COVID-19 vaccine could contribute to kidney injury, drawing from my personal experience and laboratory findings.

Spike Protein and the Kidney

The COVID-19 mRNA vaccines work by instructing the body to produce the SARS-CoV-2 spike protein. This protein is designed to train the immune system to recognize the virus. However, emerging evidence, the spike protein can travel through the bloodstream and interact with other organs.

In my case, laboratory tests revealed abnormally high levels of spike protein even years after vaccination. These proteins bind to ACE2 receptors, which are located throughout the body, including the small blood vessels of the kidneys—particularly in the glomerulus, the kidney’s filtering unit.

The Role of ACE2 in Kidney Function

ACE2 is an enzyme that helps regulate blood pressure and maintain kidney health by converting Angiotensin II (a powerful vasoconstrictor) into Angiotensin 1-7, which relaxes blood vessels and protects the kidneys. When the spike protein binds to ACE2, it reduces the receptor’s activity.

This disruption prevents Angiotensin II from being converted efficiently into Angiotensin 1-7, leading to Angiotensin II accumulation. High levels of Angiotensin II cause:

Vasoconstriction – the narrowing of small blood vessels
Increased intraglomerular pressure – elevated pressure within the kidney’s filtering units
Inflammation and oxidative stress – damaging kidney cells over time

From Injury to Kidney Failure

When the glomerulus is exposed to sustained high pressure and reduced blood flow, it gradually loses its filtering ability. Over time, this can progress to chronic kidney injury, and eventually end-stage renal disease, which requires dialysis to sustain life.

This explains why, since 2020, there has been a noticeable number of relatively young individuals suddenly ending up on dialysis—far earlier than would be expected based solely on diabetes, hypertension, or age-related kidney decline.

Why Awareness Matters

Understanding the potential kidney effects of the spike protein is crucial for early detection and prevention. Regular monitoring of kidney function—through serum creatinine, urine tests, and blood pressure tracking—is vital, especially for those experiencing unusual fatigue, swelling, or changes in urination after vaccination.