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Spike Proteins and the Liver: Unraveling Hidden Risks

Emerging research indicates that SARS-CoV-2 spike proteins can directly affect liver function, potentially increasing intrahepatic pressure and disrupting hepatocyte activity. Studies suggest that spike proteins may trigger inflammatory cascades and impair albumin production, compromising liver synthetic capacity. The discovery of reverse transcriptase activity in human cells raises concerns about prolonged spike protein expression from viral RNA fragments. Clinically, these effects may manifest as fluid retention, edema, or elevated liver enzymes, highlighting an underappreciated risk of liver stress. Understanding these mechanisms is critical for evaluating long-term organ health in patients exposed to spike proteins. Tags: #SpikeProtein #LiverHealth #IntrahepaticPressure #Albumin #ReverseTranscriptase #HepatocyteFunction #COVID19Research #LiverStress #ViralRNA #PostViralEffects

Glenn Rosaroso Vale, MT(AMT), MS(IT), MBA

8/30/20252 min read

The Spike Protein and the Liver Connection

The SARS-CoV-2 pandemic has not only transformed global health perspectives but has also ignited a multitude of scientific inquiries concerning the relationship between viral components, particularly the spike protein, and vital organs such as the liver. This discourse is critical, as the liver is rich in ACE2 receptors, making it a susceptible target for circulating spike proteins, whether derived from infection or vaccine-induced expression.

Understanding the Role of ACE2 Receptors

ACE2 receptors play a significant role in the body, primarily in the regulation of blood pressure and inflammation. However, their interaction with spike proteins poses an underexplored risk. Initially, it was assumed that these proteins merely facilitated the entry of the virus into cells; recent research indicates a more complex interaction could be at play. Studies suggest that the spike protein, after binding to ACE2 receptors, may lead to cellular dysfunction, potentially disrupting liver function.

Potential Implications for Liver Health

The implications of the spike protein’s interaction with liver cells could be profound. Emerging evidence suggests the possibility of genomic interactions, where the spike protein may influence the genetic machinery within liver cells, potentially leading to inflammation and cellular apoptosis. Liver dysfunction can manifest in various clinical symptoms, including jaundice, fatigue, and altered metabolic processes, which may complicate health outcomes—particularly in individuals with preexisting liver conditions.

Understanding the full spectrum of risks posed by spike proteins is essential. Ongoing research is necessary to elucidate the molecular pathways involved in these interactions and to assess their long-term impacts on liver health. As scientists continue to study the spike protein’s effects, health professionals must remain vigilant and consider these risks when evaluating patients presenting with symptoms indicative of underlying liver issues.

In conclusion, as the world navigates the aftermath of the pandemic, the connection between spike proteins and liver health warrants significant attention. Delving deeper into this relationship can help researchers better inform public health strategies and interventions to safeguard liver function during and after the pandemic. In my case, it has been five years since my COVID-19 infection, and my intrahepatic pressure remains elevated. My antibody level against the spike protein is greater than 2,500, suggesting that the vaccine, rather than the virus itself, is more likely contributing to this liver issue.